Gut microbes shape microglia and cognitive function during malnutrition

dc.contributor.authorHan, Jun
dc.contributor.authorBauer, Kylynda C.
dc.contributor.authorYork, Elisa M.
dc.contributor.authorCirstea, Mihai S.
dc.contributor.authorRadisavljevic, Nina
dc.contributor.authorPetersen, Charisse
dc.contributor.authorHuus, Kelsey E.
dc.contributor.authorBrown, Eric M.
dc.contributor.authorBozorgmehr, Tahereh
dc.contributor.authorBerdun, Rebeca
dc.contributor.authorBernier, Louis-Philippe
dc.contributor.authorLee, Amy H. Y.
dc.contributor.authorWoodward, Sarah E.
dc.contributor.authorKrekhno, Zakhar
dc.contributor.authorHancock, Robert E. W.
dc.contributor.authorAyala, Victoria
dc.contributor.authorMacVicar, Brian A.
dc.contributor.authorFinlay, Barton Brett
dc.date.accessioned2022-02-23T23:06:50Z
dc.date.available2022-02-23T23:06:50Z
dc.date.copyright2022en_US
dc.date.issued2022-01-12
dc.description.abstractFecal-oral contamination promotes malnutrition pathology. Lasting consequences of early life malnutrition include cognitive impairment, but the underlying pathology and influence of gut microbes remain largely unknown. Here, we utilize an established murine model combining malnutrition and iterative exposure to fecal commensals (MAL-BG). The MAL-BG model was analyzed in comparison to malnourished (MAL mice) and healthy (CON mice) controls. Malnourished mice display poor spatial memory and learning plasticity, as well as altered microglia, non-neuronal CNS cells that regulate neuroimmune responses and brain plasticity. Chronic fecal-oral exposures shaped microglial morphology and transcriptional profile, promoting phagocytic features in MAL-BG mice. Unexpectedly, these changes occurred independently from significant cytokine-induced inflammation or blood–brain barrier (BBB) disruption, key gut-brain pathways. Metabolomic profiling of the MAL-BG cortex revealed altered polyunsaturated fatty acid (PUFA) profiles and systemic lipoxidative stress. In contrast, supplementation with an ω3 PUFA/antioxidant-associated diet (PAO) mitigated cognitive deficits within the MAL-BG model. These findings provide valued insight into the malnourished gut microbiota-brain axis, highlighting PUFA metabolism as a potential therapeutic target.en_US
dc.description.reviewstatusRevieweden_US
dc.description.scholarlevelFacultyen_US
dc.description.sponsorshipBill and Melinda Gates Foundation Canadian Institute for Advanced Research Canadian Institutes of Health Research. Grant Number: FDN-159935 Leducq Foundation Departament de Salut, Generalitat de Catalunyaen_US
dc.identifier.citationBauer, K. C., York, E. M., Cirstea, M. S., Radisavljevic, N., Petersen, C., Huus, K. E., Brown, E. M., Bozorgmehr, T., Berdún, R., Bernier, L.-P., Lee, A. H. Y., Woodward, S. E., Krekhno, Z., Han, J., Hancock, R. E. W., Ayala, V., MacVicar, B. A., & Finlay, B. B. (2022). Gut microbes shape microglia and cognitive function during malnutrition. Glia, 1– 22. https://doi.org/10.1002/glia.24139en_US
dc.identifier.urihttps://doi.org/10.1002/glia.24139
dc.identifier.urihttp://hdl.handle.net/1828/13752
dc.language.isoenen_US
dc.publisherGLIAen_US
dc.subjectbehavior
dc.subjectgut-brain axis
dc.subjectmalnutrition
dc.subjectmicrobiome
dc.subjectmicroglia
dc.subjectneurometabolism
dc.subjectThe Metabolomics Innovation Centre
dc.subject.departmentDivision of Medical Sciences
dc.subject.departmentSchool of Medical Sciences
dc.titleGut microbes shape microglia and cognitive function during malnutritionen_US
dc.typeArticleen_US

Files

Original bundle
Now showing 1 - 1 of 1
Loading...
Thumbnail Image
Name:
Han_Jun_GLIA_2022.pdf
Size:
4.89 MB
Format:
Adobe Portable Document Format
Description:
License bundle
Now showing 1 - 1 of 1
No Thumbnail Available
Name:
license.txt
Size:
2 KB
Format:
Item-specific license agreed upon to submission
Description: